Hedin, K E <br/><br/>
Gi proteins use a novel beta gamma- and Ras-independent pathway to activate extracellular signal-regulated kinase and mobilize AP-1 transcription factors in Jurkat T lymphocytes.  [electronic resource] 

 -  The Journal of biological chemistry  Jul 1999 
 - 19992-20001 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.274.28.19992  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Benzoquinones<br/>Calcium-Calmodulin-Dependent Protein Kinases--metabolism<br/>Cyclic AMP-Dependent Protein Kinases--metabolism<br/>DNA-Binding Proteins<br/>Enzyme Activation<br/>GTP-Binding Proteins--metabolism<br/>Gene Expression Regulation<br/>Humans<br/>Jurkat Cells<br/>Lactams, Macrocyclic<br/>MAP Kinase Kinase 1<br/>Mitogen-Activated Protein Kinase Kinases<br/>Pertussis Toxin<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Protein Serine-Threonine Kinases--metabolism<br/>Protein-Tyrosine Kinases--metabolism<br/>Proto-Oncogene Proteins--metabolism<br/>Proto-Oncogene Proteins c-fos--metabolism<br/>Quinones--pharmacology<br/>RNA, Messenger--metabolism<br/>Receptors, Opioid, delta--metabolism<br/>Rifabutin--analogs & derivatives<br/>Signal Transduction<br/>Transcription Factor AP-1--metabolism<br/>Transcription Factors<br/>Virulence Factors, Bordetella--pharmacology<br/>beta-Adrenergic Receptor Kinases<br/>ets-Domain Protein Elk-1<br/>ras Proteins--metabolism