Kompa, A R <br/><br/>
Desensitization of cardiac beta-adrenoceptor signaling with heart failure produced by myocardial infarction in the rat. Evidence for the role of Gi but not Gs or phosphorylating proteins.  [electronic resource] 

 -  Journal of molecular and cellular cardiology  Jun 1999 
 - 1185-201 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-2828 
<br/><br/><label>Standard No.: </label>10.1006/jmcc.1999.0951  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adrenergic beta-Agonists--pharmacology<br/>Animals<br/>Arrestins--drug effects<br/>Autoradiography<br/>Blood Pressure<br/>Calcium--pharmacology<br/>Catechols--pharmacology<br/>Colforsin--pharmacology<br/>Cyclic AMP-Dependent Protein Kinases--drug effects<br/>G-Protein-Coupled Receptor Kinase 2<br/>GTP-Binding Protein alpha Subunits, Gi-Go--drug effects<br/>GTP-Binding Protein alpha Subunits, Gs--drug effects<br/>Gene Expression Regulation<br/>Heart Atria--drug effects<br/>Heart Failure--metabolism<br/>Heart Ventricles--drug effects<br/>Isoproterenol--pharmacology<br/>Myocardial Infarction--metabolism<br/>Organ Size<br/>Pertussis Toxin<br/>Phosphorylation<br/>Propanolamines--pharmacology<br/>RNA, Messenger<br/>Rats<br/>Receptors, Adrenergic, beta--metabolism<br/>Signal Transduction<br/>Virulence Factors, Bordetella--pharmacology<br/>beta-Adrenergic Receptor Kinases<br/>beta-Arrestins