Chen, G <br/><br/>
Dissociation of CDK2 from cyclin A in response to the topoisomerase II inhibitor etoposide in v-src-transformed but not normal NIH 3T3 cells.  [electronic resource] 

 -  Experimental cell research  Jun 1999 
 - 327-36 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0014-4827 
<br/><br/><label>Standard No.: </label>10.1006/excr.1999.4484  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells--drug effects<br/>Animals<br/>CDC2 Protein Kinase--metabolism<br/>CDC2-CDC28 Kinases<br/>Cell Cycle--drug effects<br/>Cell Transformation, Viral<br/>Cyclin A--metabolism<br/>Cyclin-Dependent Kinase 2<br/>Cyclin-Dependent Kinase 4<br/>Cyclin-Dependent Kinases--metabolism<br/>Enzyme Activation--drug effects<br/>Etoposide--pharmacology<br/>Genes, src--physiology<br/>Mice<br/>Oncogene Protein pp60(v-src)--genetics<br/>Protein Serine-Threonine Kinases--metabolism<br/>Proto-Oncogene Proteins<br/>Topoisomerase II Inhibitors