Rousseau, D <br/><br/>
Growth inhibition by CDK-cyclin and PCNA binding domains of p21 occurs by distinct mechanisms and is regulated by ubiquitin-proteasome pathway.  [electronic resource] 

 -  Oncogene  May 1999 
 - 3290-302 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0950-9232 
<br/><br/><label>Standard No.: </label>10.1038/sj.onc.1202681  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells<br/>Animals<br/>Binding Sites<br/>CDC2-CDC28 Kinases<br/>Carrier Proteins<br/>Cell Cycle Proteins<br/>Cell Division<br/>Cyclin-Dependent Kinase 2<br/>Cyclin-Dependent Kinase Inhibitor p21<br/>Cyclin-Dependent Kinases--metabolism<br/>Cyclins--genetics<br/>Cysteine Endopeptidases--metabolism<br/>DNA-Binding Proteins<br/>E2F Transcription Factors<br/>Genetic Vectors<br/>Humans<br/>Mice<br/>Multienzyme Complexes--metabolism<br/>Mutagenesis<br/>Proliferating Cell Nuclear Antigen--metabolism<br/>Proteasome Endopeptidase Complex<br/>Protein Serine-Threonine Kinases--metabolism<br/>Recombinant Fusion Proteins--genetics<br/>Retinoblastoma-Binding Protein 1<br/>S Phase<br/>Transcription Factor DP1<br/>Transcription Factors--metabolism<br/>Transcription, Genetic<br/>Tumor Cells, Cultured<br/>Ubiquitins--metabolism