Li, J J <br/><br/>
Overexpression and amplification of c-myc in the Syrian hamster kidney during estrogen carcinogenesis: a probable critical role in neoplastic transformation.  [electronic resource] 

 -  Cancer research  May 1999 
 - 2340-6 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0008-5472 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aneuploidy<br/>Animals<br/>Carcinoma, Renal Cell--chemically induced<br/>Cell Transformation, Neoplastic--chemically induced<br/>Chromosome Mapping<br/>Cocarcinogenesis<br/>Cricetinae<br/>Diethylstilbestrol--toxicity<br/>Estrogens<br/>Gene Amplification<br/>Gene Expression Regulation--drug effects<br/>Gene Expression Regulation, Neoplastic--drug effects<br/>Genes, fos<br/>Genes, myc<br/>In Situ Hybridization<br/>Karyotyping<br/>Kidney--drug effects<br/>Kidney Cortex--drug effects<br/>Kidney Medulla--drug effects<br/>Kidney Neoplasms--chemically induced<br/>Male<br/>Mesocricetus--genetics<br/>Neoplasms, Hormone-Dependent--chemically induced<br/>Orchiectomy<br/>Proliferating Cell Nuclear Antigen--analysis<br/>RNA, Messenger--biosynthesis<br/>RNA, Neoplasm--biosynthesis<br/>Receptors, Estrogen--drug effects<br/>S Phase<br/>Species Specificity