Kramer, A A <br/><br/>
Induction of tolerance to hemorrhagic or endotoxic shock involves activation of NF-kappaB.  [electronic resource] 

 -  The Journal of surgical research  May 1999 
 - 89-94 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-4804 
<br/><br/><label>Standard No.: </label>10.1006/jsre.1999.5571  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Nucleus--metabolism<br/>Cells, Cultured<br/>Cytosol--metabolism<br/>DNA-Binding Proteins--metabolism<br/>Hemorrhage--physiopathology<br/>I-kappa B Proteins<br/>Lipopolysaccharides--pharmacology<br/>Macrophages, Peritoneal--drug effects<br/>Male<br/>NF-KappaB Inhibitor alpha<br/>NF-kappa B--antagonists & inhibitors<br/>RNA, Messenger--genetics<br/>Rats<br/>Rats, Sprague-Dawley<br/>Shock, Hemorrhagic--physiopathology<br/>Shock, Septic--physiopathology<br/>Signal Transduction<br/>Survival<br/>Transcription, Genetic--drug effects<br/>Tumor Necrosis Factor-alpha--genetics