Brar, B K <br/><br/>
Heat shock proteins delivered with a virus vector can protect cardiac cells against apoptosis as well as against thermal or hypoxic stress.  [electronic resource] 

 -  Journal of molecular and cellular cardiology  Jan 1999 
 - 135-46 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-2828 
<br/><br/><label>Standard No.: </label>10.1006/jmcc.1998.0857  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Annexin A5--analysis<br/>Apoptosis<br/>Blotting, Western<br/>Cell Hypoxia<br/>Cell Line<br/>Cell Survival<br/>Cells, Cultured<br/>Ceramides--pharmacology<br/>Green Fluorescent Proteins<br/>HSP70 Heat-Shock Proteins--genetics<br/>Heart--physiology<br/>Heat-Shock Proteins--genetics<br/>Immunophilins--metabolism<br/>In Situ Nick-End Labeling<br/>Luminescent Proteins<br/>Rats<br/>Rats, Sprague-Dawley<br/>Simplexvirus--metabolism<br/>Tacrolimus Binding Proteins<br/>Temperature<br/>Time Factors