Genetic dissection of SLE pathogenesis: adoptive transfer of Sle1 mediates the loss of tolerance by bone marrow-derived B cells. [electronic resource]
Producer: 19990413Description: 2415-21 p. digitalISSN:- 0022-1767
- Adoptive Transfer
- Animals
- Antigens, CD -- biosynthesis
- Antigens, Differentiation, T-Lymphocyte -- biosynthesis
- Autoantibodies -- biosynthesis
- Autoantigens -- biosynthesis
- B-Lymphocyte Subsets -- immunology
- B7-2 Antigen
- Bone Marrow Cells -- immunology
- Bone Marrow Transplantation -- immunology
- CD4-Positive T-Lymphocytes -- transplantation
- Gene Expression Regulation -- immunology
- Genetic Markers -- immunology
- Immune Tolerance -- genetics
- Immunoglobulin G -- biosynthesis
- Immunophenotyping
- Lectins, C-Type
- Lupus Erythematosus, Systemic -- etiology
- Membrane Glycoproteins -- biosynthesis
- Mice
- Mice, Congenic
- Mice, Inbred C57BL
- Mice, Inbred NZB
- Nucleosomes -- immunology
- T-Lymphocyte Subsets -- metabolism
- Transplantation Chimera -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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