Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death. [electronic resource]
Producer: 19990205Description: 1156-63 p. digitalISSN:- 0021-9258
- Amino Acid Sequence
- Antigens, CD -- metabolism
- Apoptosis
- BH3 Interacting Domain Death Agonist Protein
- Carrier Proteins -- chemistry
- Caspases -- metabolism
- Cycloheximide -- pharmacology
- Cytochrome c Group -- metabolism
- Cytosol -- metabolism
- Hydrolysis
- Mitochondria -- drug effects
- Molecular Sequence Data
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Receptors, Tumor Necrosis Factor -- metabolism
- Receptors, Tumor Necrosis Factor, Type I
- Tumor Necrosis Factor-alpha -- pharmacology
- bcl-X Protein
- fas Receptor -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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