A K+ channel splice variant common in human heart lacks a C-terminal domain required for expression of rapidly activating delayed rectifier current. [electronic resource]
Producer: 19981106Description: 27231-5 p. digitalISSN:- 0021-9258
- Alternative Splicing
- Cation Transport Proteins
- Cloning, Molecular
- DNA-Binding Proteins
- ERG1 Potassium Channel
- Electric Conductivity
- Ether-A-Go-Go Potassium Channels
- Humans
- Long QT Syndrome -- metabolism
- Molecular Sequence Data
- Myocardium -- metabolism
- Potassium -- metabolism
- Potassium Channels -- genetics
- Potassium Channels, Voltage-Gated
- Recombinant Proteins -- metabolism
- Sequence Analysis, DNA
- Sequence Deletion
- Trans-Activators
- Transcriptional Regulator ERG
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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