Overexpression of the Na(+)-Ca2+ exchanger leads to enhanced inotropic responsiveness to Na(+)-channel agonist without sarcoplasmic reticulum protein changes in transgenic mice. [electronic resource]
Producer: 19981002Description: 786-90 p. digitalISSN:- 0006-291X
- Animals
- Azetidines -- pharmacology
- Calcium-Binding Proteins -- metabolism
- Calcium-Transporting ATPases -- metabolism
- Calsequestrin -- metabolism
- Dogs
- Gene Expression
- Immunohistochemistry
- In Vitro Techniques
- Isoproterenol -- pharmacology
- Mice
- Mice, Inbred C3H
- Mice, Inbred C57BL
- Mice, Transgenic
- Myocardial Contraction -- drug effects
- Proteins -- metabolism
- Sarcoplasmic Reticulum -- metabolism
- Sodium Channel Agonists
- Sodium-Calcium Exchanger -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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