Restoration of growth arrest by p16INK4, p21WAF1, pRB, and p53 is dependent on the integrity of the endogenous cell-cycle control pathways in human glioblastoma cell lines. [electronic resource]
Producer: 19980223Description: 51-62 p. digitalISSN:- 0014-4827
- Carrier Proteins -- biosynthesis
- Cell Cycle -- physiology
- Cell Cycle Proteins
- Cell Division
- Cyclin-Dependent Kinase Inhibitor p15
- Cyclin-Dependent Kinase Inhibitor p16
- Cyclin-Dependent Kinase Inhibitor p21
- Cyclins -- biosynthesis
- Enzyme Inhibitors -- metabolism
- Glioblastoma
- Humans
- Kinetics
- Recombinant Proteins -- metabolism
- Retinoblastoma Protein -- biosynthesis
- Retroviridae
- Transfection
- Tumor Cells, Cultured
- Tumor Suppressor Protein p53 -- biosynthesis
- Tumor Suppressor Proteins
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
There are no comments on this title.
Log in to your account to post a comment.