Tumor necrosis factor-alpha promotes sustained cyclooxygenase-2 expression: attenuation by dexamethasone and NSAIDs. [electronic resource]
Producer: 19980225Description: 727-43 p. digitalISSN:- 0090-6980
- Amnion -- cytology
- Anti-Inflammatory Agents, Non-Steroidal -- pharmacology
- Cells, Cultured
- Cyclooxygenase 1
- Cyclooxygenase 2
- Dexamethasone -- pharmacology
- Dinoprostone -- biosynthesis
- Dose-Response Relationship, Drug
- Glucocorticoids -- pharmacology
- Humans
- Isoenzymes -- drug effects
- Membrane Proteins
- Prostaglandin-Endoperoxide Synthases -- drug effects
- RNA, Messenger -- biosynthesis
- Tumor Necrosis Factor-alpha -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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