Inhibition of protein tyrosine kinases attenuates increases in expression of transforming growth factor-beta isoforms and their receptors following arterial injury. [electronic resource]
Producer: 19980122Description: 2461-70 p. digitalISSN:- 1079-5642
- Activin Receptors, Type I
- Angioplasty, Balloon -- adverse effects
- Animals
- Antigens, CD -- biosynthesis
- Carotid Arteries -- metabolism
- Carotid Artery Injuries
- Cells, Cultured
- Enzyme Inhibitors -- pharmacology
- Gene Expression Regulation -- drug effects
- Genistein -- pharmacology
- Integrin alphaV
- Integrin beta3
- Isoflavones -- pharmacology
- Male
- Muscle, Smooth, Vascular -- drug effects
- Platelet Membrane Glycoproteins -- biosynthesis
- Protein Serine-Threonine Kinases -- biosynthesis
- Protein-Tyrosine Kinases -- antagonists & inhibitors
- RNA, Messenger -- biosynthesis
- Rats
- Rats, Sprague-Dawley
- Receptor, Transforming Growth Factor-beta Type I
- Receptor, Transforming Growth Factor-beta Type II
- Receptors, Growth Factor -- biosynthesis
- Receptors, Transforming Growth Factor beta -- biosynthesis
- Signal Transduction
- Transforming Growth Factor beta -- biosynthesis
- Tunica Intima -- injuries
- Wound Healing -- physiology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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