APA

Byers P. H., Duvic M., Atkinson M., Robinow M., Smith L. T., Krane S. M., Greally M. T., Ludman M., Matalon R., Pauker S., Quanbeck D. & Schwarze U. (19971020). Ehlers-Danlos syndrome type VIIA and VIIB result from splice-junction mutations or genomic deletions that involve exon 6 in the COL1A1 and COL1A2 genes of type I collagen. : American journal of medical genetics.

Chicago

Byers P H, Duvic M, Atkinson M, Robinow M, Smith L T, Krane S M, Greally M T, Ludman M, Matalon R, Pauker S, Quanbeck D and Schwarze U. 19971020. Ehlers-Danlos syndrome type VIIA and VIIB result from splice-junction mutations or genomic deletions that involve exon 6 in the COL1A1 and COL1A2 genes of type I collagen. : American journal of medical genetics.

Harvard

Byers P. H., Duvic M., Atkinson M., Robinow M., Smith L. T., Krane S. M., Greally M. T., Ludman M., Matalon R., Pauker S., Quanbeck D. and Schwarze U. (19971020). Ehlers-Danlos syndrome type VIIA and VIIB result from splice-junction mutations or genomic deletions that involve exon 6 in the COL1A1 and COL1A2 genes of type I collagen. : American journal of medical genetics.

MLA

Byers P H, Duvic M, Atkinson M, Robinow M, Smith L T, Krane S M, Greally M T, Ludman M, Matalon R, Pauker S, Quanbeck D and Schwarze U. Ehlers-Danlos syndrome type VIIA and VIIB result from splice-junction mutations or genomic deletions that involve exon 6 in the COL1A1 and COL1A2 genes of type I collagen. : American journal of medical genetics. 19971020.