Point mutations can inactivate in vitro and in vivo activities of p16(INK4a)/CDKN2A in human glioma. [electronic resource]
Producer: 19970307Description: 603-9 p. digitalISSN:- 0950-9232
- Alleles
- Carrier Proteins -- genetics
- Cell Cycle
- Chromosome Deletion
- Chromosomes, Human, Pair 9
- Cyclin-Dependent Kinase Inhibitor p16
- Enzyme Inhibitors -- metabolism
- Genes, Tumor Suppressor
- Glioma -- enzymology
- Humans
- Phosphorylation
- Protein Biosynthesis
- Protein Kinase Inhibitors
- Recombinant Proteins -- biosynthesis
- Retinoblastoma Protein -- metabolism
- Transcription, Genetic
- Transfection
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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