Gap junction turnover, intracellular trafficking, and phosphorylation of connexin43 in brefeldin A-treated rat mammary tumor cells. [electronic resource]
Producer: 19960122Description: 1193-203 p. digitalISSN:- 0021-9525
- Animals
- Antifungal Agents -- pharmacology
- Brefeldin A
- Cell Line
- Connexin 43 -- antagonists & inhibitors
- Cycloheximide -- pharmacology
- Cyclopentanes -- pharmacology
- Endoplasmic Reticulum -- metabolism
- Gap Junctions -- drug effects
- Golgi Apparatus -- metabolism
- Isomerases -- metabolism
- Kinetics
- Mammary Neoplasms, Animal
- Phosphorylation
- Protein Biosynthesis
- Protein Disulfide-Isomerases
- Protein Synthesis Inhibitors -- pharmacology
- Rats
- Receptors, Cell Surface
- Receptors, Fibroblast Growth Factor
- Sialoglycoproteins -- metabolism
- Tumor Cells, Cultured
- Uncoupling Agents -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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