Amyloid precursor protein in the cerebral cortex is rapidly and persistently induced by loss of subcortical innervation. [electronic resource]
Producer: 19931020Description: 8712-6 p. digitalISSN:- 0027-8424
- 5,7-Dihydroxytryptamine -- toxicity
- Acetylcholine -- metabolism
- Actins -- biosynthesis
- Afferent Pathways -- drug effects
- Amyloid beta-Protein Precursor -- biosynthesis
- Animals
- Cerebral Cortex -- drug effects
- Choline O-Acetyltransferase -- metabolism
- Diabetes Mellitus, Experimental -- metabolism
- Functional Laterality
- Glucocorticoids -- pharmacology
- Kinetics
- Lidocaine -- pharmacology
- Male
- N-Methylaspartate -- toxicity
- Norepinephrine -- physiology
- Oxidopamine -- toxicity
- Physostigmine -- pharmacology
- Polyribosomes -- metabolism
- RNA, Messenger -- analysis
- Raphe Nuclei -- drug effects
- Rats
- Rats, Sprague-Dawley
- Substantia Innominata -- drug effects
- Time Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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