High-dose cyclophosphamide inhibits anterior chamber-associated immune deviation (ACAID) and the production of extracellular antigen-specific T cell proteins induced by trinitrophenylated (TNP) spleen cells. [electronic resource]
Producer: 19951031Description: 284-8 p. digitalISSN:- 0008-8749
- Animals
- Anterior Chamber -- immunology
- Cyclophosphamide -- pharmacology
- Dose-Response Relationship, Drug
- Female
- Immune Tolerance -- drug effects
- Immunosuppressive Agents -- pharmacology
- Mice
- Mice, Inbred BALB C
- Rabbits
- Receptors, Antigen, T-Cell, alpha-beta -- biosynthesis
- Spleen -- metabolism
- Trinitrobenzenes -- immunology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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