Thyrotropin-releasing hormone stimulates MAP kinase activity in GH3 cells by divergent pathways. Evidence of a role for early tyrosine phosphorylation. [electronic resource]
Producer: 19940318Description: 3783-8 p. digitalISSN:- 0021-9258
- Adaptor Proteins, Signal Transducing
- Adaptor Proteins, Vesicular Transport
- Adenosine Triphosphate -- metabolism
- Animals
- Calcium-Calmodulin-Dependent Protein Kinases -- metabolism
- Cell Line
- Epidermal Growth Factor -- pharmacology
- Genes, ras -- drug effects
- Kinetics
- Phosphoproteins -- isolation & purification
- Phosphorylation
- Phosphotyrosine
- Pituitary Neoplasms
- Protein Kinase C -- antagonists & inhibitors
- Protein Serine-Threonine Kinases -- metabolism
- Proteins -- metabolism
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-raf
- Proto-Oncogene Proteins p21(ras) -- metabolism
- Proto-Oncogenes -- drug effects
- Rats
- Shc Signaling Adaptor Proteins
- Src Homology 2 Domain-Containing, Transforming Protein 1
- Tetradecanoylphorbol Acetate -- pharmacology
- Thyrotropin-Releasing Hormone -- pharmacology
- Tumor Cells, Cultured
- Tyrosine -- analogs & derivatives
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Publication Type: Journal Article
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