Disruption of the EGFR-SQSTM1 interaction by a stapled peptide suppresses lung cancer via activating autophagy and inhibiting EGFR signaling. [electronic resource]
Producer: 20201215Description: 23-35 p. digitalISSN:- 1872-7980
- Animals
- Antineoplastic Agents -- pharmacology
- Apoptosis
- Autophagy
- Biomarkers, Tumor -- genetics
- Carcinoma, Non-Small-Cell Lung -- drug therapy
- Cell Proliferation
- Drug Resistance, Neoplasm
- ErbB Receptors -- antagonists & inhibitors
- Gene Expression Regulation, Neoplastic
- Humans
- Lung Neoplasms -- drug therapy
- Male
- Mice
- Mice, Inbred BALB C
- Mice, Nude
- Mutation
- Peptide Fragments -- pharmacology
- Protein Kinase Inhibitors -- pharmacology
- Protein Multimerization
- Sequestosome-1 Protein -- antagonists & inhibitors
- Signal Transduction
- Tumor Cells, Cultured
- Xenograft Model Antitumor Assays
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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