MELK promotes Endometrial carcinoma progression via activating mTOR signaling pathway. [electronic resource]
Producer: 20200930Description: 102609 p. digitalISSN:- 2352-3964
- Animals
- Cell Line, Tumor
- Cell Movement -- drug effects
- Cell Proliferation -- drug effects
- Cohort Studies
- Disease Progression
- E2F1 Transcription Factor -- metabolism
- Endometrial Neoplasms -- genetics
- Female
- Gene Expression Regulation, Neoplastic -- drug effects
- Gene Knockdown Techniques
- Humans
- Mechanistic Target of Rapamycin Complex 1 -- metabolism
- Mechanistic Target of Rapamycin Complex 2 -- metabolism
- Mice, Inbred BALB C
- Mice, Nude
- Middle Aged
- Models, Biological
- Multivariate Analysis
- Naphthyridines -- pharmacology
- Prognosis
- Protein Binding -- drug effects
- Protein Serine-Threonine Kinases -- metabolism
- Signal Transduction -- drug effects
- TOR Serine-Threonine Kinases -- metabolism
- Transcriptional Activation -- drug effects
- mTOR Associated Protein, LST8 Homolog -- metabolism
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Publication Type: Journal Article
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