TLR4 induced Wnt3a-Dvl3 restrains the intensity of inflammation and protects against endotoxin-driven organ failure through GSK3β/β-catenin signaling. [electronic resource]
Producer: 20200406Description: 153-164 p. digitalISSN:- 1872-9142
- Animals
- Cytokines -- metabolism
- Dishevelled Proteins -- metabolism
- Endotoxins -- metabolism
- Glycogen Synthase Kinase 3 beta -- metabolism
- Humans
- Inflammation -- metabolism
- Lipopolysaccharides -- pharmacology
- Mice
- Mice, Inbred C57BL
- Monocytes -- metabolism
- Multiple Organ Failure -- metabolism
- Phosphorylation -- physiology
- Signal Transduction -- physiology
- Toll-Like Receptor 4 -- metabolism
- Transcription Factor RelA -- metabolism
- Tumor Necrosis Factor-alpha -- metabolism
- Wnt3A Protein -- metabolism
- beta Catenin -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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