Attenuation of secondary damage and Aβ deposits in the ipsilateral thalamus of dMCAO rats through reduction of cathepsin B by bis(propyl)-cognitin, a multifunctional dimer. [electronic resource]
Producer: 20210106Description: 107786 p. digitalISSN:- 1873-7064
- Amyloid beta-Peptides -- drug effects
- Animals
- Antigens, CD -- metabolism
- Antigens, Differentiation, Myelomonocytic -- metabolism
- Antigens, Nuclear -- metabolism
- Cathepsin B -- antagonists & inhibitors
- Dipeptides -- pharmacology
- Disease Models, Animal
- GABA-A Receptor Antagonists -- pharmacology
- Glial Fibrillary Acidic Protein -- metabolism
- Gliosis -- metabolism
- Infarction, Middle Cerebral Artery -- metabolism
- Nerve Tissue Proteins -- metabolism
- Neuroglia -- drug effects
- Neurons -- drug effects
- Neuroprotective Agents -- pharmacology
- Rats
- Tacrine -- analogs & derivatives
- Thalamus -- drug effects
- Ventral Thalamic Nuclei -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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