Diastolic dysfunction is initiated by cardiomyocyte impairment ahead of endothelial dysfunction due to increased oxidative stress and inflammation in an experimental prediabetes model. [electronic resource]
Producer: 20200709Description: 119-131 p. digitalISSN:- 1095-8584
- Actin Cytoskeleton -- metabolism
- Animals
- Connectin -- metabolism
- Cytokines -- metabolism
- Diastole
- Disease Models, Animal
- Endothelium, Vascular -- pathology
- Guanylate Cyclase -- metabolism
- Heart Ventricles -- drug effects
- Hydrogen Peroxide -- metabolism
- Inflammation -- pathology
- Male
- Multienzyme Complexes -- metabolism
- Myocytes, Cardiac -- drug effects
- Myosins -- metabolism
- NADH, NADPH Oxidoreductases -- metabolism
- Nitric Oxide -- pharmacology
- Nitric Oxide Synthase Type III -- metabolism
- Oxidative Stress
- Peptides -- metabolism
- Phosphorylation
- Prediabetic State -- pathology
- Rats, Wistar
- Superoxides -- metabolism
- Vasodilation -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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