Syk-dependent glycolytic reprogramming in dendritic cells regulates IL-1β production to β-glucan ligands in a TLR-independent manner. [electronic resource]
Producer: 20200603Description: 1325-1335 p. digitalISSN:- 1938-3673
- Animals
- Dendritic Cells -- immunology
- Glycolysis
- Interleukin-1beta -- biosynthesis
- Lectins, C-Type -- metabolism
- Ligands
- Mice
- Myeloid Differentiation Factor 88 -- metabolism
- NLR Family, Pyrin Domain-Containing 3 Protein -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Protein Serine-Threonine Kinases -- metabolism
- Proto-Oncogene Proteins c-akt -- metabolism
- Signal Transduction
- Syk Kinase -- genetics
- Toll-Like Receptors -- metabolism
- beta-Glucans -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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