Augmented O-GlcNAcylation attenuates intermittent hypoxia-induced cardiac remodeling through the suppression of NFAT and NF-κB activities in mice. [electronic resource]
Producer: 20201104Description: 1858-1871 p. digitalISSN:- 1348-4214
- Acylation
- Animals
- Cell Line
- Diabetes Mellitus, Type 2 -- complications
- Echocardiography
- Glycogen Synthase Kinase 3 beta -- metabolism
- HEK293 Cells
- Humans
- Hypoxia -- pathology
- Mice
- Mice, Transgenic
- Myocardium -- metabolism
- N-Acetylglucosaminyltransferases -- genetics
- NF-kappa B -- metabolism
- NFATC Transcription Factors -- metabolism
- Oxidative Stress
- Phosphorylation
- Ventricular Remodeling
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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