Targeting the inhibition of fatty acid amide hydrolase ameliorate the endocannabinoid-mediated synaptic dysfunction in a valproic acid-induced rat model of Autism. [electronic resource]
Producer: 20210106Description: 107736 p. digitalISSN:- 1873-7064
- Amidohydrolases -- antagonists & inhibitors
- Animals
- Arachidonic Acids -- metabolism
- Autism Spectrum Disorder -- chemically induced
- Benzamides -- pharmacology
- Carbamates -- pharmacology
- Disease Models, Animal
- Endocannabinoids -- metabolism
- Enzyme Inhibitors -- toxicity
- Female
- Long-Term Synaptic Depression -- drug effects
- Neuronal Plasticity -- drug effects
- Polyunsaturated Alkamides -- metabolism
- Prefrontal Cortex -- metabolism
- Pregnancy
- Prenatal Exposure Delayed Effects -- chemically induced
- Protein Transport -- drug effects
- Rats
- Receptors, AMPA -- drug effects
- Receptors, Metabotropic Glutamate -- drug effects
- Social Behavior
- Valproic Acid -- toxicity
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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