Titin truncations lead to impaired cardiomyocyte autophagy and mitochondrial function in vivo. [electronic resource]
Producer: 20200206Description: 1971-1981 p. digitalISSN:- 1460-2083
- Acetylation
- Animals
- Autophagosomes -- metabolism
- Autophagy -- genetics
- Cardiomyopathy, Dilated -- genetics
- Cathepsin B -- metabolism
- Cells, Cultured
- Connectin -- genetics
- Male
- Microtubule-Associated Proteins -- metabolism
- Mitochondria, Heart -- metabolism
- Mitochondrial Proteins -- metabolism
- Myocytes, Cardiac -- metabolism
- NAD -- analogs & derivatives
- Rats
- Reactive Oxygen Species -- metabolism
- Sequence Deletion
- Sequestosome-1 Protein -- metabolism
- TOR Serine-Threonine Kinases -- metabolism
- Ubiquitination
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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