APA
Czech J., Cordua S., Weinbergerova B., Baumeister J., Crepcia A., Han L., Maié T., Costa I. G., Denecke B., Maurer A., Schubert C., Feldberg K., Gezer D., Brümmendorf T. H., Müller-Newen G., Mayer J., Racil Z., Kubesova B., Knudsen T., Sørensen A. L., Holmström M., Kjær L., Skov V., Larsen T. S., Hasselbalch H. C., Chatain N. & Koschmieder S. (20190809). JAK2V617F but not CALR mutations confer increased molecular responses to interferon-α via JAK1/STAT1 activation. : Leukemia.
Chicago
Czech Julia, Cordua Sabrina, Weinbergerova Barbora, Baumeister Julian, Crepcia Assja, Han Lijuan, Maié Tiago, Costa Ivan G, Denecke Bernd, Maurer Angela, Schubert Claudia, Feldberg Kristina, Gezer Deniz, Brümmendorf Tim H, Müller-Newen Gerhard, Mayer Jiri, Racil Zdenek, Kubesova Blanka, Knudsen Trine, Sørensen Anders L, Holmström Morten, Kjær Lasse, Skov Vibe, Larsen Thomas Stauffer, Hasselbalch Hans C, Chatain Nicolas and Koschmieder Steffen. 20190809. JAK2V617F but not CALR mutations confer increased molecular responses to interferon-α via JAK1/STAT1 activation. : Leukemia.
Harvard
Czech J., Cordua S., Weinbergerova B., Baumeister J., Crepcia A., Han L., Maié T., Costa I. G., Denecke B., Maurer A., Schubert C., Feldberg K., Gezer D., Brümmendorf T. H., Müller-Newen G., Mayer J., Racil Z., Kubesova B., Knudsen T., Sørensen A. L., Holmström M., Kjær L., Skov V., Larsen T. S., Hasselbalch H. C., Chatain N. and Koschmieder S. (20190809). JAK2V617F but not CALR mutations confer increased molecular responses to interferon-α via JAK1/STAT1 activation. : Leukemia.
MLA
Czech Julia, Cordua Sabrina, Weinbergerova Barbora, Baumeister Julian, Crepcia Assja, Han Lijuan, Maié Tiago, Costa Ivan G, Denecke Bernd, Maurer Angela, Schubert Claudia, Feldberg Kristina, Gezer Deniz, Brümmendorf Tim H, Müller-Newen Gerhard, Mayer Jiri, Racil Zdenek, Kubesova Blanka, Knudsen Trine, Sørensen Anders L, Holmström Morten, Kjær Lasse, Skov Vibe, Larsen Thomas Stauffer, Hasselbalch Hans C, Chatain Nicolas and Koschmieder Steffen. JAK2V617F but not CALR mutations confer increased molecular responses to interferon-α via JAK1/STAT1 activation. : Leukemia. 20190809.