β-Arrestin2 Inhibits Expression of Inflammatory Cytokines in BEAS-2B Lung Epithelial Cells Treated with Cigarette Smoke Condensate via Inhibition of Autophagy. [electronic resource]
Producer: 20181109Description: 1270-1285 p. digitalISSN:- 1421-9778
- AMP-Activated Protein Kinases -- metabolism
- Autophagy -- drug effects
- Autophagy-Related Proteins -- metabolism
- Cell Line
- Chemokine CCL2 -- metabolism
- Epithelial Cells -- cytology
- Humans
- Interleukin-6 -- metabolism
- Lung -- metabolism
- Microscopy, Confocal
- Propranolol -- pharmacology
- RNA Interference
- RNA, Small Interfering -- metabolism
- Signal Transduction -- drug effects
- Sirolimus -- pharmacology
- Smoke
- TOR Serine-Threonine Kinases -- metabolism
- Nicotiana -- chemistry
- beta-Arrestin 2 -- antagonists & inhibitors
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Publication Type: Journal Article
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