Coronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk. [electronic resource]
Producer: 20190215Description: e1007681 p. digitalISSN:- 1553-7404
- Basic Helix-Loop-Helix Transcription Factors -- genetics
- Binding Sites
- Cell Differentiation -- genetics
- Coronary Artery Disease -- genetics
- Epistasis, Genetic
- Genetic Predisposition to Disease
- Genome-Wide Association Study
- Humans
- Muscle, Smooth, Vascular -- metabolism
- Polymorphism, Single Nucleotide
- Primary Cell Culture
- Signal Transduction
- Smad3 Protein -- genetics
- Transforming Growth Factor beta -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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