A dominant dendrite phenotype caused by the disease-associated G253D mutation in doublecortin (DCX) is not due to its endocytosis defect. [electronic resource]
Producer: 20190417Description: 18890-18902 p. digitalISSN:- 1083-351X
- Adaptor Protein Complex 2 -- metabolism
- Animals
- Binding Sites
- COS Cells
- Cell Adhesion Molecules -- metabolism
- Chlorocebus aethiops
- Dendrites -- genetics
- Doublecortin Domain Proteins
- Doublecortin Protein
- Endocytosis -- genetics
- HEK293 Cells
- Humans
- Mice
- Microtubule-Associated Proteins -- genetics
- Mutation
- Nerve Growth Factors -- metabolism
- Neurons -- cytology
- Neuropeptides -- genetics
- Rats
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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