Inhibition of sonic hedgehog and PI3K/Akt/mTOR pathways cooperate in suppressing survival, self-renewal and tumorigenic potential of glioblastoma-initiating cells. [electronic resource]
Producer: 20190419Description: 11-23 p. digitalISSN:- 1573-4919
- Animals
- Antineoplastic Agents -- pharmacology
- Biphenyl Compounds -- pharmacology
- Cell Proliferation
- Glioblastoma -- drug therapy
- Hedgehog Proteins -- metabolism
- Humans
- Imidazoles -- pharmacology
- Mice
- Mice, SCID
- Neoplastic Stem Cells -- drug effects
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphoinositide-3 Kinase Inhibitors
- Proto-Oncogene Proteins c-akt -- metabolism
- Pyridines -- pharmacology
- Quinolines -- pharmacology
- Signal Transduction -- drug effects
- Smoothened Receptor -- antagonists & inhibitors
- TOR Serine-Threonine Kinases -- antagonists & inhibitors
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Publication Type: Journal Article
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