Von Hippel-Lindau Protein Is Required for Optimal Alveolar Macrophage Terminal Differentiation, Self-Renewal, and Function. [electronic resource]
Producer: 20191125Description: 1738-1746 p. digitalISSN:- 2211-1247
- Animals
- Antigens, Differentiation, Myelomonocytic -- genetics
- Basic Helix-Loop-Helix Transcription Factors -- genetics
- CD11 Antigens -- genetics
- CD11b Antigen -- genetics
- Cell Differentiation -- genetics
- Cell Proliferation -- genetics
- Cytokine Receptor Common beta Subunit -- deficiency
- Gene Deletion
- Gene Expression Regulation
- Humans
- Hypoxia -- genetics
- Hypoxia-Inducible Factor 1, alpha Subunit -- genetics
- Lung -- metabolism
- Macrophages, Alveolar -- metabolism
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Oxygen -- pharmacology
- Receptors, IgG -- genetics
- Sialic Acid Binding Immunoglobulin-like Lectins
- Signal Transduction
- Von Hippel-Lindau Tumor Suppressor Protein -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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