Overexpression of endophilin A1 exacerbates synaptic alterations in a mouse model of Alzheimer's disease. [electronic resource]
Producer: 20190102Description: 2968 p. digitalISSN:- 2041-1723
- Adaptor Proteins, Signal Transducing -- genetics
- Adenosine Triphosphate -- metabolism
- Alzheimer Disease -- genetics
- Amyloid beta-Peptides -- genetics
- Amyloid beta-Protein Precursor -- genetics
- Animals
- Animals, Genetically Modified
- Antioxidants -- metabolism
- Crosses, Genetic
- Disease Models, Animal
- Gene Expression Regulation
- Hippocampus -- metabolism
- Humans
- Long-Term Potentiation
- Mice
- Mice, Transgenic
- Mitochondria -- metabolism
- Neurons -- metabolism
- Neurotransmitter Agents -- metabolism
- Peptide Fragments -- metabolism
- Reactive Oxygen Species -- metabolism
- Synapses -- metabolism
- Synaptic Vesicles -- metabolism
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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