The tumour suppressor OPCML promotes AXL inactivation by the phosphatase PTPRG in ovarian cancer. [electronic resource]
Producer: 20190513ISSN:- 1469-3178
- Animals
- Benzocycloheptenes -- pharmacology
- Cell Adhesion Molecules -- metabolism
- Cell Line, Tumor
- Cell Movement -- drug effects
- Cell Survival -- drug effects
- Chickens
- Cholesterol -- metabolism
- Enzyme Activation -- drug effects
- Epithelial Cells -- drug effects
- Fallopian Tubes -- pathology
- Female
- GPI-Linked Proteins -- metabolism
- Gene Silencing -- drug effects
- Humans
- Intercellular Signaling Peptides and Proteins -- metabolism
- MAP Kinase Signaling System -- drug effects
- Membrane Microdomains -- metabolism
- Neoplasm Invasiveness
- Ovarian Neoplasms -- metabolism
- Phosphorylation -- drug effects
- Protein Binding -- drug effects
- Proto-Oncogene Proteins -- metabolism
- Receptor Protein-Tyrosine Kinases -- metabolism
- Receptor-Like Protein Tyrosine Phosphatases, Class 5 -- metabolism
- Treatment Outcome
- Triazoles -- pharmacology
- Tumor Suppressor Proteins -- metabolism
- Axl Receptor Tyrosine Kinase
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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