Elevation of the TP53 isoform Δ133p53β in glioblastomas: an alternative to mutant p53 in promoting tumor development. [electronic resource]
Producer: 20191022Description: 77-88 p. digitalISSN:- 1096-9896
- Animals
- Antigens, CD -- metabolism
- Antigens, Differentiation, Myelomonocytic -- metabolism
- Antineoplastic Agents, Alkylating -- pharmacology
- B7-H1 Antigen -- metabolism
- Biomarkers, Tumor -- genetics
- Brain Neoplasms -- drug therapy
- Cell Line
- Chemokine CCL2 -- metabolism
- Drug Resistance, Neoplasm
- Gene Expression Regulation, Neoplastic
- Glioblastoma -- drug therapy
- Humans
- Macrophages -- metabolism
- Mice
- Mutation
- Oxidative Stress
- Protein Isoforms
- Receptors, Cell Surface -- metabolism
- Receptors, Granulocyte-Macrophage Colony-Stimulating Factor -- metabolism
- Signal Transduction
- Temozolomide -- pharmacology
- Tumor Hypoxia
- Tumor Microenvironment
- Tumor Suppressor Protein p53 -- genetics
- Up-Regulation
- CD163 Antigen
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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