Perindopril, fosinopril and losartan inhibited the progression of diethylnitrosamine-induced hepatocellular carcinoma in mice via the inactivation of nuclear transcription factor kappa-B. [electronic resource]
Producer: 20180827Description: 32-40 p. digitalISSN:- 1879-3169
- Angiotensin II Type 1 Receptor Blockers -- pharmacology
- Angiotensin-Converting Enzyme Inhibitors -- pharmacology
- Animals
- Antineoplastic Agents -- pharmacology
- Carcinoma, Hepatocellular -- chemically induced
- Cyclin D1 -- genetics
- Diethylnitrosamine
- Fosinopril -- pharmacology
- Liver Neoplasms, Experimental -- chemically induced
- Losartan -- pharmacology
- Male
- Matrix Metalloproteinase 2 -- metabolism
- Mice
- NF-KappaB Inhibitor alpha -- metabolism
- NF-kappa B -- metabolism
- Niacinamide -- analogs & derivatives
- Perindopril -- pharmacology
- Phenylurea Compounds -- pharmacology
- Phosphorylation
- Renin-Angiotensin System -- drug effects
- Signal Transduction -- drug effects
- Sorafenib
- Time Factors
- Transcription Factor RelA -- metabolism
- Transforming Growth Factor beta1 -- metabolism
- Tumor Necrosis Factor-alpha -- metabolism
- Vascular Endothelial Growth Factor A -- metabolism
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Publication Type: Comparative Study; Journal Article
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