Targeted complement inhibition salvages stressed neurons and inhibits neuroinflammation after stroke in mice. [electronic resource]
Producer: 20190920ISSN:- 1946-6242
- Animals
- Brain Ischemia -- complications
- Complement Activation
- Complement System Proteins -- metabolism
- Disease Models, Animal
- Disease Susceptibility
- Immunoglobulin M -- metabolism
- Infarction, Middle Cerebral Artery -- complications
- Inflammation -- complications
- Ischemia -- pathology
- Mice
- Mice, Inbred C57BL
- Microglia -- metabolism
- Nerve Degeneration -- complications
- Neurons -- drug effects
- Neuroprotective Agents -- pharmacology
- Opsonin Proteins -- metabolism
- Phagocytosis -- drug effects
- Pneumonia -- complications
- Proto-Oncogene Proteins c-fos -- metabolism
- Recovery of Function -- drug effects
- Stroke -- complications
- Time Factors
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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