Impaired proteoglycan glycosylation, elevated TGF-β signaling, and abnormal osteoblast differentiation as the basis for bone fragility in a mouse model for gerodermia osteodysplastica. [electronic resource]
Producer: 20180710Description: e1007242 p. digitalISSN:- 1553-7404
- Animals
- Bone Diseases -- congenital
- Cell Differentiation
- Decorin -- metabolism
- Dermatan Sulfate -- metabolism
- Disease Models, Animal
- Dwarfism -- metabolism
- Female
- Fractures, Bone -- genetics
- Glycosylation
- Golgi Matrix Proteins
- Mesenchymal Stem Cells -- pathology
- Mice, Inbred C57BL
- Mice, Transgenic
- Osteoblasts -- metabolism
- Proteoglycans -- metabolism
- Signal Transduction
- Skin Diseases, Genetic -- metabolism
- Transforming Growth Factor beta -- metabolism
- Vesicular Transport Proteins -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.