Hyper-acetylation contributes to the sensitivity of chemo-resistant prostate cancer cells to histone deacetylase inhibitor Trichostatin A. [electronic resource]
Producer: 20191015Description: 1909-1922 p. digitalISSN:- 1582-4934
- Acetyl Coenzyme A -- metabolism
- Adaptor Proteins, Signal Transducing
- Allografts
- Animals
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- Carrier Proteins -- genetics
- Cell Cycle Proteins
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- Cisplatin -- pharmacology
- Docetaxel -- pharmacology
- Doxorubicin -- pharmacology
- Drug Resistance, Neoplasm -- drug effects
- Endoplasmic Reticulum Stress -- drug effects
- Eukaryotic Initiation Factors
- Gene Expression Regulation, Neoplastic
- Histone Deacetylase Inhibitors -- pharmacology
- Histones -- genetics
- Humans
- Hydroxamic Acids -- pharmacology
- Male
- Mice
- Mice, Inbred C57BL
- Phosphoproteins -- genetics
- Prostatic Neoplasms -- drug therapy
- Protein Isoforms -- genetics
- Proto-Oncogene Proteins c-akt -- genetics
- Signal Transduction
- TOR Serine-Threonine Kinases -- genetics
- Tumor Burden -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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