The Mammalian Target of Rapamycin and DNA methyltransferase 1 axis mediates vascular endothelial dysfunction in response to disturbed flow. [electronic resource]
Producer: 20190718Description: 14996 p. digitalISSN:- 2045-2322
- Animals
- Arteries -- pathology
- Atherosclerosis -- genetics
- Cattle
- Connective Tissue Growth Factor -- genetics
- Cyclin A -- genetics
- DNA (Cytosine-5-)-Methyltransferase 1 -- metabolism
- DNA Methylation -- physiology
- Disease Models, Animal
- Endothelium, Vascular -- pathology
- Epigenesis, Genetic -- physiology
- Hemorheology -- physiology
- Human Umbilical Vein Endothelial Cells
- Humans
- Mice
- Mice, Inbred C57BL
- Mice, Knockout, ApoE
- Promoter Regions, Genetic -- genetics
- TOR Serine-Threonine Kinases -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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