Suppression of Trim32 Enhances Motor Function Repair after Traumatic Brain Injury Associated with Antiapoptosis. [electronic resource]
Producer: 20180514Description: 1276-1285 p. digitalISSN:- 1555-3892
- Animals
- Apoptosis -- genetics
- Behavior, Animal
- Brain Injuries, Traumatic -- genetics
- DNA-Binding Proteins
- Disease Models, Animal
- Electron Transport Complex IV -- metabolism
- In Situ Nick-End Labeling
- Male
- Mice, Knockout
- Motor Activity
- Nerve Regeneration
- Nerve Tissue Proteins -- metabolism
- Nuclear Proteins -- metabolism
- Rats, Sprague-Dawley
- Transcription Factors -- genetics
- Tripartite Motif Proteins -- genetics
- Tumor Protein p73 -- metabolism
- Ubiquitin-Protein Ligases -- genetics
- Up-Regulation -- genetics
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Publication Type: Journal Article
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