Dickkopf 3 (Dkk3) Improves Amyloid-β Pathology, Cognitive Dysfunction, and Cerebral Glucose Metabolism in a Transgenic Mouse Model of Alzheimer's Disease. [electronic resource]
Producer: 20180409Description: 733-746 p. digitalISSN:- 1875-8908
- Adaptor Proteins, Signal Transducing
- Aged, 80 and over
- Alzheimer Disease -- complications
- Amyloid beta-Peptides -- metabolism
- Amyloid beta-Protein Precursor -- genetics
- Animals
- Cerebral Cortex -- metabolism
- Cognition Disorders -- diagnostic imaging
- Disease Models, Animal
- Exploratory Behavior -- physiology
- Female
- Gene Expression Regulation -- genetics
- Glucose -- metabolism
- Humans
- Intercellular Signaling Peptides and Proteins -- genetics
- Male
- Maze Learning -- physiology
- Mice
- Mice, Transgenic
- Mutation -- genetics
- Presenilin-1 -- genetics
- Wnt Signaling Pathway -- genetics
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.