Recombinant human IgG1 based Fc multimers, with limited FcR binding capacity, can effectively inhibit complement-mediated disease. [electronic resource]
Producer: 20180531Description: 97-108 p. digitalISSN:- 1095-9157
- Animals
- Cells, Cultured
- Complement C1q -- immunology
- Complement C3 -- metabolism
- Complement System Proteins -- immunology
- Cytotoxicity, Immunologic
- Disease Models, Animal
- Erythrocytes -- physiology
- Glomerulonephritis, Membranous
- Hemolysis
- Humans
- Immune System Diseases -- immunology
- Immunoglobulin Fc Fragments -- genetics
- Immunoglobulin G -- genetics
- Molecular Targeted Therapy
- Protein Binding
- Protein Multimerization
- Receptors, Fc -- metabolism
- Thy-1 Antigens -- immunology
- Transgenes -- genetics
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.