Releasing Syntaphilin Removes Stressed Mitochondria from Axons Independent of Mitophagy under Pathophysiological Conditions. [electronic resource]
Producer: 20170727Description: 595-610.e6 p. digitalISSN:- 1097-4199
- Aged
- Aged, 80 and over
- Alzheimer Disease -- genetics
- Amyotrophic Lateral Sclerosis -- genetics
- Animals
- Axons -- metabolism
- Calcium -- metabolism
- Carrier Proteins -- metabolism
- Cerebral Cortex -- cytology
- Endosomes -- metabolism
- Female
- Humans
- Male
- Membrane Proteins
- Mice
- Mice, Knockout
- Microscopy, Immunoelectron
- Microtubule-Associated Proteins -- metabolism
- Middle Aged
- Mitochondria -- metabolism
- Mitophagy -- genetics
- Motor Neurons -- metabolism
- Mutation
- Nerve Tissue Proteins -- metabolism
- Neurons -- cytology
- Rats
- Spinal Cord -- cytology
- Stress, Physiological
- Ubiquitin-Protein Ligases -- genetics
- Vesicular Transport Proteins -- metabolism
No physical items for this record
Publication Type: Journal Article; Video-Audio Media
There are no comments on this title.
Log in to your account to post a comment.