Salinomycin acts through reducing AKT-dependent thymidylate synthase expression to enhance erlotinib-induced cytotoxicity in human lung cancer cells. [electronic resource]
Producer: 20171003Description: 59-66 p. digitalISSN:- 1090-2422
- Antineoplastic Agents -- pharmacology
- Carcinoma, Non-Small-Cell Lung -- drug therapy
- Cell Line, Tumor
- ErbB Receptors -- metabolism
- Erlotinib Hydrochloride -- pharmacology
- Humans
- Lung Neoplasms -- drug therapy
- Proto-Oncogene Proteins c-akt -- metabolism
- Pyrans -- pharmacology
- Thymidylate Synthase -- metabolism
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Publication Type: Journal Article
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