Antagonism of EGFR and Notch limits resistance to EGFR inhibitors and radiation by decreasing tumor-initiating cell frequency. [electronic resource]
Producer: 20171218ISSN:- 1946-6242
- Aldehyde Dehydrogenase -- metabolism
- Animals
- Antibodies -- pharmacology
- Antineoplastic Agents -- pharmacology
- Carcinogenesis -- pathology
- Carcinoma, Non-Small-Cell Lung -- drug therapy
- Cell Count
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- Drug Resistance, Neoplasm -- drug effects
- Epithelial-Mesenchymal Transition -- drug effects
- ErbB Receptors -- antagonists & inhibitors
- Lung Neoplasms -- drug therapy
- Mice, SCID
- Molecular Targeted Therapy
- Neoplasm Recurrence, Local -- drug therapy
- Neoplastic Stem Cells -- drug effects
- Protein Kinase Inhibitors -- pharmacology
- Radiation Tolerance -- drug effects
- Receptors, Notch -- antagonists & inhibitors
- Signal Transduction -- drug effects
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Publication Type: Journal Article
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