β-Arrestin-biased AT1R stimulation promotes extracellular matrix synthesis in renal fibrosis. [electronic resource]
Producer: 20170918Description: F1-F8 p. digitalISSN:- 1522-1466
- Angiotensin II -- analogs & derivatives
- Angiotensin II Type 1 Receptor Blockers -- pharmacology
- Animals
- Benzimidazoles -- pharmacology
- Biphenyl Compounds
- Cell Line
- Collagen Type I -- metabolism
- Disease Models, Animal
- Extracellular Matrix -- metabolism
- Fibroblasts -- drug effects
- Fibronectins -- metabolism
- Fibrosis
- Kidney -- drug effects
- Kidney Diseases -- etiology
- Male
- Mice, Inbred C57BL
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 3 -- metabolism
- Phosphorylation
- RNA Interference
- Rats
- Receptor, Angiotensin, Type 1 -- drug effects
- Signal Transduction
- Tetrazoles -- pharmacology
- Time Factors
- Transfection
- Ureteral Obstruction -- complications
- beta-Arrestin 1 -- agonists
- beta-Arrestin 2 -- agonists
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Publication Type: Journal Article
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